The basis for supporting this theory is as follows:
1. Geist (1933) used a capillary with a diameter equal to 1 / 3 of the inner diameter of the stroma to suck out the endometrial cells in the blood from the interstitial part of the fallopian tube, which confirmed that the endometrial cells could flow through the fallopian tube;
2. Ridley (1938) confirmed that there were free and active endometriosis fragments in the fallopian tube;
3. Te Linde and Wharton (1953) inverted the uterus of rhesus monkey and made menstruation flow into the pelvic cavity. It was confirmed that endometriosis occurred after the interval of February to two years. Scott and the Linde (1954) confirmed the growth ability of endometrium after implantation, and hughesdon (1958) further confirmed it.
4. During the exploratory laparotomy in the menstrual period, it can be seen that there is blood flow from the fimbrial end of the fallopian tube, which contains active endometrial cells; any factors that hinder the smooth flow of blood flow, such as imperforate hymen, vaginal atresia, cervical stenosis or adhesion of the cervix and uterine cavity, are prone to suffer from endometriosis.
5. Most of the ectopic lesions are distributed in the free parts of the pelvic cavity, such as the uterine rectum lacuna, ovarian fossa, and so on. Because the ovary is close to the oviduct fimbria, it is also easy to implant.
The theory can explain the ectopia in the basin, abdominal cavity, genitalia, and surgical incision, but it can not explain the ectopia outside the basin, abdominal cavity, head, umbilical cord and lymphatics, and can not explain why 70% - 90% of women have menstrual blood reflux, but only a few have endometriosis, so other theories are put forward on the basis of this.
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